In our review, we found most frequently increased CCL2 (41.7% in vitro, 45.5% in vivo, and 33.3% in human serum during the acute phase of infection), CCL5 (50% in vitro, 38.6% in vivo), and CXCL10 (41.7% in vitro, 40.9% in vivo, and 33.3% in human serum during the acute phase of infection). This evidence concerns the gene CXCL10 and infection.