We hypothesized that exposure to blast will (1) induce increased bacterial diversity in blood; (2) induce alterations in the levels of gut permeability protein biomarkers in circulation including Zonulin, LBP, Claudin-3, and I-FABP; and (3) that these molecular alterations related to IP will be associated with acute blast-related exposure symptomology. This evidence concerns the gene FABP2 and incontinentia pigmenti.