In the oxidative T98G cell line, the heightened acetylation of signaling pathways components, such as those involved in epidermal growth factor receptor (EGFR) signaling pathways—mitogen-activated protein kinase (MAPK), forkhead box O (FoxO), and the phosphoinositide 3-kinase (PI3K-Akt)—highlights acetylation’s pivotal role in regulating key cellular functions, including proliferation and response to treatment in glioblastoma [27,28]. The gene discussed is AKT1; the disease is glioblastoma.