Thus, haematological oncologies such as high-grade serous ovarian cancer (HGSOC), epithelial ovarian cancer (EOC) and other ovarian cancer subtypes have several SE-associated pathological mechanisms, namely BRD4 and SE copy-number amplification similar to breast cancer [111]; ovarian cancer-like shared mechanisms of cisplatin-induced SE activation (concluded based on elevated seRNA expression); and BRD4 amplification. This evidence concerns the gene BRD4 and ovarian carcinoma.