The existence of data indicating a 3–6 times higher relative risk of venous thrombosis and the impact on the occurrence of recurrent venous thrombosis in individuals with FVIII values above 1.5 U/mL (over 150%), as well as the role of FVIII in the pathogenesis of atherothrombosis, highlights the significance of these findings in inducing a prothrombotic state, atherothrombotic complications, and venous thromboembolism in patients with hyperthyroidism [16,17]. This evidence concerns the gene F8 and venous thromboembolism.