CD163 and idiopathic pulmonary fibrosis: Furthermore, detailed analysis showed that CD163/LGMN-Mφ was one of the dominant populations of the recently recruited monocyte-derived macrophages during the first four weeks of COVID-19 ARDS, alongside FCN1-Mono and Mono-Mφ; SARS-CoV2 may induce CD163/LGMN-Mφ macrophage population phenotypes that strongly resemble the ones of IPF-specific macrophages [16].