The same research group reported that the EO induces the suppression of lipid accumulation in differentiated 3T3-L1 adipocytes, as well as a decrease in the expression of PPARγ, CEBPα (CCAAT enhancer-binding protein alpha, a transcriptor regulator involved among others in adipogenesis [244]), FABP (fatty acid-binding protein 4, with a recognized role in the development of insulin resistance and atherosclerosis [245]), and GPDH (a key enzyme involved in triglyceride synthesis [246]); they also confirmed an anti-obesity effect of the EO in a rat model [247]. The gene discussed is CEBPA; the disease is Insulin resistance.