For instance, mutated variants of the UV excision repair protein (RAD23B) were linked to the emergence of primary lung cancer regardless of patient ethnicity, and RPS6 was associated with drug resistance in non-small cell lung cancer, whereas the protein ARPC5 was found to upregulate a tumor suppressor agent in lung squamous cell carcinoma [56,57,58]. This evidence concerns the gene ARPC5 and neoplasm.