UCA1 and serous adenocarcinoma: Besides, Wambecke et al. revealed that UCA1 was responsible for enhancing DDP resistance to both DDP-resistant OAW42 (OAW42/DDP) cells and their parental line, whereas UCA1 silencing induced a S-G2/M phase block in the former cells and increased apoptosis at G1 phase in DDP-sensitive OAW42 and high-grade serous adenocarcinoma OVCAR3 cells.