Although none of the quantified alpha (Adra1a, Adra1b, Adra1) and beta (Adrb1, Adrb2) adrenergic receptors were regulated (Figure 4), the inward sodium transporters Scn1b and Scn5a were over-expressed, presumably to compensate for the low sodium level: this might be relevant in the treatment of Brugada syndrome [92]. Here, SCN5A is linked to Brugada syndrome.