FST and acute kidney injury: Furthermore, when compared to the levels in healthy controls, a substantial increase in urinary follistatin was evident in cases of sepsis-associated AKI (n = 20, 1214 ± 783.4) and drug-induced nephropathy (n = 27, 514.9 ± 246.4), while no such elevation was observed in AKI attributed to hypotension (n = 11, 268.6 ± 207.8), CIN (n = 10, 179.6 ± 90.6) or surgery (n = 3, 402.1 ± 291.7) (Figure 2D).