Since inflammation is supposed to play a role in IUGR, Kirici et al. recently studied pro-inflammatory markers (erythrocyte sedimentation rate, sensitivity C-reactive protein, interleukin-6, and tumor necrosis factor-alpha) and found them to be increased, while the anti-inflammatory factor interleukin-10 was decreased in the maternal serum of IUGR pregnancies. This evidence concerns the gene IL10 and fetal growth restriction.