Considering that macrophages are a majority cell type for IFN-β secretion in the injured hearts, we resuspended MICFs infected with MGT in solution containing neutralizing antibody (NAb) targeting IFNAR, which can prevent IFN-β binding to IFNAR receptors, and transplanted the MICFs into the infarction zone of the hearts during MI. The gene discussed is IFNAR1; the disease is myocardial infarction.