Discussed earlier, damaged epithelium releases a number of soluble mediators promoting remodelling and inflammation (e.g., TSLP, IL25, and IL33), and are not only highly expressed in asthmatic airways, but represent genetic loci identified in a number of genome-wide association (GWA) studies correlating with asthma susceptibility (Cookson, 2004; Allakhverdi et al., 2007; Grotenboer et al., 2013; Moheimani et al., 2016). Here, TSLP is linked to asthma.