MTMR3 and preeclampsia: In turn, the increase of miR-181a reduced MTMR3, inhibited the occurrence of autophagy, prevented protein aggregation in trophoblasts and abnormal placental dysfunction, and provided a potential therapeutic target for the diagnosis of preeclampsia (36), thereby further reducing the risk of cardiovascular disease, diabetes and other metabolic diseases in women and infants who survived preeclampsia (37).