Using real-time quantitative PCR, we further showed that AT alleviated lipodystrophy-induced activation of hepatic lipogenesis (Pparγ, Fas, and Scd1) and inhibition of triglyceride-rich lipoprotein packaging (Mtp) and fatty acid β-oxidation (Pparα and Cpt1α) in Seipin/Apoe dKO mice (Fig. 2F). Here, CPT1A is linked to lipodystrophy.