Conversely, other studies have observed that tumor-infiltrating natural killer (NK) cells can recruit cDC1s through production of CCL5 and XC-chemokine ligand 1 (XCL1) (82) and promote their survival with FMS-related tyrosine kinase 3 ligand (FLT3l) (83). The gene discussed is FLT3LG; the disease is neoplasm.