CD79A and metabolic dysfunction-associated steatotic liver disease: Long-term inflammation and fibrosis in both human and mouse models of MASLD were linked to liver-resident IgA producing cells expressing PDL1, hindering body’s ability to effectively avoid cancer.103,104 In a mouse model of MASH, B cells within the liver stimulated by microbial factors from the hut, contributed to liver inflammation and fibrosis.105