Furthermore, we monitored the expression of AARS1, YAP, and TEAD1 during N-methyl-N-nitrosourea–induced (MNU-induced) mouse GC progression and found that expression levels of AARS1 and lactylation levels of YAP-TEAD1 were obviously increased upon MNU treatment (Figure 5B). The gene discussed is TEAD1; the disease is gastric cancer.