In line with this, transient expression of both MYC and Cyclin T1 by a single intramyocardial dose of a modified RNA coding for both genes was shown to be a potential regenerative therapeutic in the heart after myocardial infarction, inducing cell cycle and division of cardiomyocytes (Figure 3G) (Boikova et al., 2023). Here, MYC is linked to myocardial infarction.