SLC9A1 and stroke disorder: To further investigate the underlying mechanisms for these phenotypic changes, and to reveal whether the selective deletion of Nhe1 in cKO brains causes uniform alterations among different microglial subpopulations, and whether it influences their functions and interactions with other cell types, we conducted scRNAseq transcriptome analysis at 3 days post-stroke timepoint to explore mechanisms between microglia-oligodendrocyte interactions.