Additionally, a differential gene expression analysis showed that S100A9 is consistently present in both SARS‐CoV‐2 and PAH samples, with a heat map indicating a high‐risk tendency propensity for S100A9 expression, implying an increased risk of PAH following SARS‐CoV‐2 infection.109. This evidence concerns the gene S100A9 and pulmonary arterial hypertension.