MYH9 and breast carcinoma: Overexpressing of TMEM120B promoted breast cancer 3D invasion, which all indicated that TMEM120B were involved in modulating mechanical force transduction.β1-integrin cycling and FA assembly were enhanced in the TMEM120B–MYH9 interaction, thus promoted translocation of TAZ into nucleus as well as subsequent elevation of phosohorylation of mTOR.