SST and early-onset autosomal dominant Alzheimer disease: In five familial Alzheimer’s disease (5 × FAD) mutation mice, EA at GV20 and GV14 ameliorates recognition learning and memory function deficits, restores glutamate decarboxylase 65 (GAD65) and somatostatin (SST) in the hippocampus, attenuates the loss of somatostatin-positive interneurons in the dorsal hippocampus, and reverses the decreased θ and γ brain oscillations (Li et al., 2023; Table 2; Figure 4).