Since disruption of aberrant lipid metabolism and subsequent LD accumulation are implicated in carcinogenesis and tumor progression, the overexpression of DGAT1 and DGAT2 in a variety of tumor cells and their regulatory roles in the formation of LDs and in the control of tumor cell growth have generated interest as to whether inhibiting DGATs can affect the growth of tumor cells [3, 23, 35–39]. Here, DGAT1 is linked to neoplasm.