In addition to decreasing the content of LDs, the DGAT1 inhibitor reduced the number and stability of the non-centrosomal microtubule-organizing center and decreased the levels of GM130, the CLIP-associated proteins 2 (CLASP2), and γ-tubulin in prostate tumor cells, thereby resulting in inhibition of cell proliferation and tumor growth [116]. The gene discussed is DGAT1; the disease is neoplasm.