Further inhibition of the bromodomain and extra-terminal protein family of histone acetylation readers suppressed the expression of perilipin 2 (PLIN2), a crucial component of LDs as well as significantly interrupted lactate-induced LD accumulation in prostate tumor cells and reduced growth and metastasis to the lungs in a prostate cancer xenograft mouse model [95]. The gene discussed is PLIN2; the disease is prostate carcinoma.