When combined with radiation, inhibition of DGAT1 by shRNA or miRNA-3918 in a xenograft mouse model of GBM U87MG radioresistance significantly attenuated tumor size and suppressed tumor growth by 65.34% and 53.64%, respectively, and prolonged survival of the mice for nearly 10 days compared with radiation alone (2 Gy, 5 times) through induction of apoptosis [141]. Here, DGAT1 is linked to glioblastoma.