For instance, fusions of oncogenic proteins TAZ, YAP1, and HIPK2 preserve their tumor-promoting function, while fusions of tumor suppressors, such as NF2, LATS1, FAT1, PTPN14, DCHS2, TAOK1, and TAOK3, results in loss of their function [49]. The gene discussed is PTPN14; the disease is neoplasm.