Unfortunately, because b cells re-activate Neurog3 under metabolic stress during diabetes development (Talchai et al., 2012), which we expect to label M−N+ progenitor-derived b cells after birth, we could not compare the function of the adult b-cell subtypes from M−N+ and M−N+ progenitors in the maternal HFD model. This evidence concerns the gene NEUROG3 and diabetes mellitus.