As previously reported, CPT1A increases reactive oxygen species in the mitochondria and promotes antioxidant defenses in PCa cells.26 CPT1A supports castration-resistant PCa by supplying acetyl groups for histone acetylation, promoting growth and antiandrogen resistance.24 Here, we confirmed the up-regulation of CPT1A in PCa. The gene discussed is CPT1A; the disease is posterior cortical atrophy.