Similarly, the severe COVID-19 group had higher levels of IgA autoantibodies to most cytokines with 18 of the 23 analytes significantly higher in the severe COVID-19 group compared to healthy controls (p ≤ 0.05; Wilcoxon–Mann–Whitney; Fig. 5b, Supplementary Fig. 3). The gene discussed is CD79A; the disease is COVID-19.