In addition, the gene expression of pro‐inflammatory cytokines (e.g., Ccl5, Cxcl9, and Cxcl10) increased in AMDs(i.v.)+light treated mice compared to PBS treated mice (Figure 7C).[30] All these phenomena suggested that AMDs‐mediated tumor‐specific PMR and lytic cell death were pro‐inflammatory. The gene discussed is CXCL10; the disease is neoplasm.