Overexpression of lncRNA H19 eliminates the anticancer effects of curcumin in lung cancer cells, whereas knockdown of H19 expression promotes curcumin-induced ferroptosis; lncRNA H19 acts as a competing endogenous RNA for miR-19b-3p, thereby enhancing the transcriptional activity of its endogenous target, ferritin heavy chain 1 (FTH1) (Zhang R. et al., 2022). Here, H19 is linked to lung cancer.