Given that neuroinflammation and immune dysregulation and their link to schizophrenia have long been discussed [25] and that there is some evidence that indicates that postnatal/childhood infections induce schizophrenia by interfering with CNS development [26–28], there may be an overlap between temporal shifts in HGF-MET signaling effects on neurodevelopment and schizophrenia. The gene discussed is MET; the disease is schizophrenia.