In addition, other study data provide evidence that the phytopharmaceutical stimulates autophagy in cancer cells through Beclin-1-independent pathways, including the modulation of Wnt/β-catenin signaling and direct inhibition of the mTOR-Unc-51-like kinase 1 pathway via ATP competition, indicating its multitarget mode of action and the importance of autophagy [56,169] (Figure 7). Here, MTOR is linked to cancer.