In ovarian cancer cells, the role of the ARHI protein (aplasia Ras homolog member I), which is a tumor suppressor, has been identified in the induction of cancer-cell dormancy through the induction of autophagy by inhibiting the phosphatidyl inositol 3-kinase (PI3K)-protein kinase B (AKT) growth-factor signaling pathway [101]. The gene discussed is AKT1; the disease is neoplasm.