Viral infection results in the activation of several pattern-recognition receptors (PRRs) [12,13] and toll-like receptors (TLRs), leading to the activation of NF-κB, a transcription factor that promotes the expression of pro-inflammatory cytokines such as interleukin 6 (IL-6) and interleukin 1β (IL-1β) [14,15,16]. The gene discussed is IL6; the disease is viral infectious disease.