AhR promotes atherosclerosis in three ways: when combined with NF-kB, it will lead to monocyte chemotaxis; AhR stimulates macrophages to absorb oxLDL to create foam cells, increasing the proliferation of vascular muscle cells and will activate NADPH oxidase which will finally lead to the production of ROS that will destroy the endothelial cells. Here, AHR is linked to atherosclerosis.