LBP and colorectal carcinoma: SNORA71A, an antisense, intron-encoded, oncogene-like snoRNA, was significantly overexpressed in CRC cells and caused increased cell proliferation, migration, and invasion while also being implicated in the LPS-induced form of the NF-κB signaling pathway through LBP, a sense-strand encoded, SNORA71A-targeted binding protein to lipopolysaccharide (LPS) [114,141,142].