The administration of propionic acid to overweight, abdominal fat, fatty liver, and insulin resistance, while significantly increasing postprandial secretion of Peptide YY or PRR (a hormone produced in the L cells of the intestinal mucosa of the ileum and colon) and Postprandial glucagon-like peptide-1 (GLP1) [66,73]. This evidence concerns the gene GCG and digestive system neoplasm.