All WSN and NS80 viruses decreased the synthesis of CX3CL1/Fractalkine, Coagulation factor III, FGF-1, and CD105/Endoglin and increased expression of Cystatin C. Chemokine CX3CL1, FGF-1, and a subpopulation of CD105+ cells are highly expressed in the convalescent phase after infection and have a neuro-regeneration property [42,43,44]. The gene discussed is CX3CL1; the disease is infection.