Yang et al. compiled the results of studies conducted on serum of MM patients and healthy controls from 2017–2021, showing the statistically significant diagnostic value of overexpression of TUG1, PCAT1, H19, HOTAIR, LINC01606, PRINS, LBX2-AS1 and reduced expression of XLOC-013703 in differentiating these two groups [48]. This evidence concerns the gene TUG1 and Miyoshi myopathy.