This is followed by an increase in the expression of cytokines that promote tumor growth, including interleukin (IL)-6, IL-10, and IL-17.34,36 Kadin et al further confirmed the presence of a Th17/Th1 phenotype in BIA-ALCL tumor lymphocytes, providing additional evidence for the potential involvement of antigenic stimulation and chronic inflammation in the initiation and promotion of BIA-ALCL.36 The gene discussed is IL10; the disease is neoplasm.