We found that 38 of the 84 RA (45.2%), 272 of the 508 SLE (53.5%), 6 of the 15 PS (40%), 5 of the 11 AS (45.4%), 156 of the 367 SS (43.3%), and 22 of the 97 MG (22.6%) patients had detectable IFN-γ auto-Abs by Gyros, which was within the range of the prevalence of IFN-γ auto-Abs detected in the general population (Figure 4, B and C). This evidence concerns the gene IFNG and rheumatoid arthritis.