Overexpression of MARCH1 promotes ovarian cancer progression through the regulation of NF-κB and Wnt/β-catenin signaling pathways [22] while MARCH1 overexpression in oral squamous cell carcinoma promotes tumor progression through regulation of the PH domain and leucine-rich repeat protein phosphatase (PHLPP) 2 [23]. This evidence concerns the gene MARCHF1 and neoplasm.