showed that the upregulated cGAS‐STING axis leads to neuroinflammation and microglial pyroptosis through NF‐κB and NLRP3 following an animal model of cerebral venous sinus thrombosis (CVST), inhibition of cGAS or downregulation of STING ameliorates neuroinflammation and microglial pyroptosis, thus improving ischemic stroke.74 This evidence concerns the gene CGAS and ischemic stroke.