Clinical data suggests that cytokine storms induced by COVID-19 involve a self-perpetuating cycle of inflammatory responses, characterized by the continuous release of pro-inflammatory cytokines, such as interleukin-1 (IL-1), interleukin-6 (IL-6), interleukin-12 (IL-12), interferon gamma (IFN-γ), and tumor necrosis factor-alpha (TNF-α), which adversely impact lung tissues11–13. Here, IFNG is linked to COVID-19.