This finding can be explained by Kaur et al[11] who elucidated that the total increase in TLC could be attributed primarily to an increase in the number of neutrophils.[79] They utilized the homeostasis model assessment of insulin resistance; which employed fasting recordings of blood glucose and insulin concentrations; as a surrogate marker for quantifying insulin resistance as well as hs-CRP (highly sensitive C reactive protein) as a marker of inflammation. Here, INS is linked to Insulin resistance.