Moreover, TYROBP and FCER1G were found to be the common hub genes of the 2 genes through the PPI network, and the common mechanism involved may be immunity and lipid metabolism.[42] TYROBP and FCER1G, important immune-related genes, are both involved in the process of atherosclerosis.[43,44] Abnormal lipid metabolism and atherosclerosis are the physiological and pathological basis of MI, of which both indicate the relationship between MI and immune mechanisms. This evidence concerns the gene TYROBP and atherosclerosis.