The platelets from GPVI-deficient mice do not tether to the sub-endothelium of injured artery walls,15 have reduced thrombus formation on collagen fibrils,16 are protected from thromboembolism,17 and protect mice from ferric chloride-induced thrombosis.18 These mice showed no increased bleeding times, suggesting that, in mice, GPVI does not play a major role in haemostasis. Here, GP6 is linked to Thromboembolism.