In our earlier investigations with bone marrow cells, we demonstrated that delivery of dsDNA fragments to experimental animals during double-strand breaks (DSB; the major intermediates in the repair of CP-induced DNA damage) repair resulted in elimination of the lymphoid lineage, which in turn led to a failure of the immune system with the consequent development of opportunistic infection, systemic inflammation, multiple organ failure, and eventual death of the animal (23). This evidence concerns the gene CP and Multiple Organ Failure.