Another in-vitro study of SARS-CoV-2 in human lung epithelium suggested that peroxisome proliferator-activated receptor alpha (PPAR-α) activation of fatty acid oxidation using fenofibrate could disrupt the SARS-CoV-2 lifecycle by blocking phospholipid accumulation and increasing glycolysis, reversing the metabolic effects of infection. This evidence concerns the gene PPARA and infection.